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In response to the imbalance of intracellular sulfur metabolism, thiol and disulfide balance, as well as the systemic regulation of Trx and Grx and the cross-talk between cell death after the occurrence of the disease, effective interventions and treatment drugs have been explored to identify pathways and multiple approaches for treating neurodegenerative diseases

Cell adhesion molecules and cytokines: After the occurrence of an ischemic stroke, there is growing evidence of post-reperfusion inflammation during which leukocytes are recruited with the help of adhesion molecules from the selectin family, integrins (1 and 2), and intercellular adhesion molecule-1 (ICAM-1) [113,119]

Prospective evaluation of the diagnostic accuracy of hepatic copper content, as determined using the entire core of a liver biopsy sample

Whether rescuing GABA-related gene defects through genetic methods can improve behavior is worthy of further exploration (102, 109)

In cells overexpressing BAG3, glutaminase (GLS) succinylation is promoted, which prevents ubiquitination and subsequent proteasomal degradation, stabilizing the GLS protein

Potential therapeutic strategies include drugs that remove iron from mitochondria (iron chelation) or drugs that neutralize free radicals (antioxidants, such as coenzyme Q10)